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Author Bashiri, F.; Hassan, C.R.C. doi  openurl
  Title Light Pollution and Its Effect on the Environment Type Journal Article
  Year 2014 Publication International Journal of Fundamental Physical Sciences Abbreviated Journal Intl. J. of Fundamental Phys. Sci.  
  Volume 4 Issue 1 Pages 8-12  
  Keywords Light pollution, human health, animal behaviour, plant growth  
  Abstract Light pollution can cause disturbance to humans as well as animals. The aim of this study is to determine the effect of light pollution on human's health, plants, animals, human body and People’s attitude about light pollution. About 90% of people strongly agreed that excessive lighting has adverse effects on a person's health. At least, 70% of people had difficulty in sleeping because of light pollution. Most of people believed that video Billboards, Spotlights, Car headlights and Street lights are the most important source of light pollution and about 60% of people agree that light pollution can affect animal’s sleep. 60% of people believed that excessive artificial light can attract several kinks of birds and insects. The results of this study indicate that the human health, plants growth and animal behaviour are strongly affected by the light pollution.‎  
  Address Faculty of Engineering University of Malaya, Kuala Lumpur, Malaysia  
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  Notes Approved no  
  Call Number IDA @ john @ Serial 313  
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Author Bedrosian, T.A. (ed) pdf  url
openurl 
  Title Circadian Disruption by Light at Night: Implications for Mood Type Book Whole
  Year 2013 Publication Abbreviated Journal  
  Volume Issue Pages  
  Keywords circadian disruption; sleep; light at night; melanopsin; mood; mental health; Mood Disorders; epigenetics; red light  
  Abstract Life on Earth has adapted to a consistent 24-h solar cycle. Circadian rhythms in physiology and behavior remain synchronized to the environment using light as the most potent entraining cue. During the past century, however, the widespread adoption of electric light has led to `round-the-clock’ societies. Instead of aligning with the environment, individuals follow artificial and often erratic light cycles created by social and work schedules. In particular, exposure to artificial light at night (LAN), termed “light pollution”, has become pervasive over the past 100 years. Virtually every individual living in the U.S. and Europe experiences this aberrant light exposure, and moreover about 20% of the population performs shift work. LAN may disrupt physiological timekeeping, leading to dysregulation of internal processes and misalignment between behavior and the environment. Recent evidence suggests that individuals exposed to excessive LAN, such as night shift workers, have increased risk for depressive disorders, but the biological mechanism remains unspecified. In mammals, intrinsically photosensitive retinal ganglion cells (ipRGCs) project light information to (1) the suprachiasmatic nucleus (SCN) in the hypothalamus, regulating circadian rhythms, and (2) to limbic regions, putatively regulating mood. Thus, LAN has the potential to affect both circadian timekeeping and mood. In this dissertation, I present evidence from rodent studies supporting the novel hypothesis that night-time exposure to light disrupts circadian organization and contributes to depressed mood. First, I consider the physiological and behavioral consequences associated with unnatural exposure to LAN. The effects of LAN on circadian output are considered in terms of locomotor activity, the diurnal cortisol rhythm, and diurnal clock protein expression in the brain in Chapter 2. The influence of LAN on behavior and brain plasticity is discussed, with particular focus on depressive-like behavior (Chapter 3) and effects of SSRI treatment (Chapter 4). Effects of LAN on structural plasticity and gene expression in the brain are described, with emphasis on potential correlates of the depressive-like behavior observed under LAN in Chapter 5. Given the prevalence of LAN exposure and its importance, strategies for reversing the effects are offered. Specifically, eliminating LAN quickly reverses behavioral and physiological effects of exposure as described in Chapter 5. In Chapter 6 I report that administration of a pharmacological cytokine inhibitor prevents depressive-like behaviors in LAN, implicating brain inflammation in the behavioral effect. Finally, I demonstrate in Chapter 7 that exposure to red wavelength LAN reduces the effects on brain and behavior, suggesting that LAN acts through specific retinal pathways involving melanopsin. Taken together, these studies demonstrate the consequences of LAN, but also outline potential avenues for prevention or intervention.  
  Address Department of Neuroscience and The Institute for Behavioral Medicine Research The Ohio State University  
  Corporate Author Thesis Ph.D. thesis  
  Publisher Place of Publication Editor Bedrosian, T.A.  
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  Call Number IDA @ john @ Serial 323  
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Author Bercz, P.A.; Jaffe, F. url  openurl
  Title Adverse health effects of shift work and shift work sleep disorder Type Journal Article
  Year 2012 Publication Dialogue and Diagnosis Abbreviated Journal  
  Volume 2 Issue Pages 13-20  
  Keywords Human Health  
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  Notes Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 506  
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Author Blask, D.; Brainard, G.; Gibbons, R.; Lockley, S.; Stevens, R.; Motta, M. openurl 
  Title Light Pollution: Adverse Health Effects of Nighttime Lighting. Type Journal Article
  Year 2012 Publication Report 4 of the Council on Science and Public Health, American Medical Association. Abbreviated Journal  
  Volume Issue Pages  
  Keywords Human Health  
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  Notes Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 508  
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Author Warthen, D.M.; Provencio, I. url  openurl
  Title The role of intrinsically photosensitive retinal ganglion cells in nonimage-forming responses to light Type Journal Article
  Year 2012 Publication Eye and Brain Abbreviated Journal  
  Volume 4 Issue Pages 43—48  
  Keywords Human Health; amygdala; bed nucleus of the stria terminalis; melanopsin; opsin; optic nerve; retina  
  Abstract Light exerts many effects on behavior and physiology. These effects can be characterized as either image-forming or nonimage-forming (NIF) visual processes. Image-forming vision refers to the process of detecting objects and organisms in the environment and distinguishing their physical characteristics, such as size, shape, and direction of motion. NIF vision, in contrast, refers to effects of light that are independent of fine spatiotemporal vision. NIF effects are many and varied, ranging from modulation of basal physiology, such as heart rate and body temperature, to changes in higher functions, such as mood and cognitive performance. In mammals, many NIF effects of light are dependent upon the inner retinal photopigment melanopsin and the cells in which melanopsin is expressed, the intrinsically photosensitive retinal ganglion cells (ipRGCs). The ipRGCs project broadly throughout the brain. Many of these projections terminate in areas known to mediate NIF effects, while others terminate in regions whose link to photoreception remains to be established. Additionally, the presence of ipRGC projections to areas of the brain with no known link to photoreception suggests the existence of additional ipRGC-mediated NIF effects. This review summarizes the known NIF effects of light and the role of melanopsin and ipRGCs in driving these effects, with an eye toward stimulating further investigation of the many and varied effects of light on physiology and behavior.  
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  Call Number LoNNe @ christopher.kyba @ Serial 519  
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