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Author Adams, C.A.; Blumenthal, A.; Fernández-Juricic, E.; Bayne, E.; St. Clair, C.C.
Title Effect of anthropogenic light on bird movement, habitat selection, and distribution: a systematic map protocol Type Journal Article
Year 2019 Publication Environmental Evidence Abbreviated Journal Environ Evid
Volume 8 Issue S1 Pages 13
Keywords Animals; BirdsDepartment of Biological Science, University of Alberta, CW 405, Biological Sciences Building, Edmonton, AB, T6G 2E9, Canada
Abstract Anthropogenic light is known or suspected to exert profound effects on many taxa, including birds. Documentation of bird aggregation around artificial light at night, as well as observations of bird reactions to strobe lights and lasers, suggests that light may both attract and repel birds, although this assumption has yet to be tested. These effects may cause immediate changes to bird movement, habitat selection and settlement, and ultimately alter bird distribution at large spatial scales. Global increases in the extent of anthropogenic light contribute to interest by wildlife managers and the public in managing light to reduce harm to birds, but there are no evidence syntheses of the multiple ways light affects birds to guide this effort. Existing reviews usually emphasize either bird aggregation or deterrence and do so for a specific context, such as aggregation at communication towers and deterrence from airports. We outline a protocol for a systematic map that collects and organizes evidence from the many contexts in which anthropogenic light is reported to affect bird movement, habitat selection, or distribution. Our map will provide an objective synthesis of the evidence that identifies subtopics that may support systematic review and knowledge gaps that could direct future research questions. These products will substantially advance an understanding of both patterns and processes associated with the responses of birds to anthropogenic light.
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ISSN 2047-2382 ISBN Medium
Area Expedition Conference
Notes Approved no
Call Number (up) GFZ @ kyba @ Serial 2547
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Author Ikeno, T.; Weil, Z.M.; Nelson, R.J.
Title Dim light at night disrupts the short-day response in Siberian hamsters Type Journal Article
Year 2014 Publication General and Comparative Endocrinology Abbreviated Journal Gen Comp Endocrinol
Volume 197 Issue Pages 56-64
Keywords 2,4-dinitro-1-flourobenzene; Dnfb; Dth; Eya3; Eyes absent 3; GnIH; GnRH; Immune function; Ld; Lps; Light pollution; Pt; Pelage; Per1; Period1; Photoperiodism; Rfrp; RFamide-related peptide; Scn; Sd; Seasonality; Tsh; TSH receptor; Tshr; dLAN; delayed-type hypersensitivity; dim light at night; gonadotropin-inhibiting hormone; gonadotropin-releasing hormone; lipopolysaccharide; long days; pars tuberalis; short days; suprachiasmatic nuclei; thyroid-stimulating hormone
Abstract Photoperiodic regulation of physiology, morphology, and behavior is crucial for many animals to survive seasonally variable conditions unfavorable for reproduction and survival. The photoperiodic response in mammals is mediated by nocturnal secretion of melatonin under the control of a circadian clock. However, artificial light at night caused by recent urbanization may disrupt the circadian clock, as well as the photoperiodic response by blunting melatonin secretion. Here we examined the effect of dim light at night (dLAN) (5lux of light during the dark phase) on locomotor activity rhythms and short-day regulation of reproduction, body mass, pelage properties, and immune responses of male Siberian hamsters. Short-day animals reduced gonadal and body mass, decreased spermatid nuclei and sperm numbers, molted to a whiter pelage, and increased pelage density compared to long-day animals. However, animals that experienced short days with dLAN did not show these short-day responses. Moreover, short-day specific immune responses were altered in dLAN conditions. The nocturnal activity pattern was blunted in dLAN hamsters, consistent with the observation that dLAN changed expression of the circadian clock gene, Period1. In addition, we demonstrated that expression levels of genes implicated in the photoperiodic response, Mel-1a melatonin receptor, Eyes absent 3, thyroid stimulating hormone receptor, gonadotropin-releasing hormone, and gonadotropin-inhibitory hormone, were higher in dLAN animals than those in short-day animals. These results suggest that dLAN disturbs the circadian clock function and affects the molecular mechanisms of the photoperiodic response.
Address Department of Neuroscience, The Ohio State University Wexner Medical Center, Columbus, OH 43210, USA. Electronic address: randy.nelson@osumc.edu
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Language English Summary Language Original Title
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Series Volume Series Issue Edition
ISSN 0016-6480 ISBN Medium
Area Expedition Conference
Notes PMID:24362257 Approved no
Call Number (up) IDA @ john @ Serial 82
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Author Reddy, A.B.; O'Neill, J.S.
Title Healthy clocks, healthy body, healthy mind Type Journal Article
Year 2010 Publication Trends in Cell Biology Abbreviated Journal Trends Cell Biol
Volume 20 Issue 1 Pages 36-44
Keywords Aging; Animals; Cell Cycle; *Circadian Rhythm; Humans; Neoplasms/genetics/metabolism; Signal Transduction
Abstract Circadian rhythms permeate mammalian biology. They are manifested in the temporal organisation of behavioural, physiological, cellular and neuronal processes. Whereas it has been shown recently that these approximately 24-hour cycles are intrinsic to the cell and persist in vitro, internal synchrony in mammals is largely governed by the hypothalamic suprachiasmatic nuclei that facilitate anticipation of, and adaptation to, the solar cycle. Our timekeeping mechanism is deeply embedded in cell function and is modelled as a network of transcriptional and/or post-translational feedback loops. Concurrent with this, we are beginning to understand how this ancient timekeeper interacts with myriad cell systems, including signal transduction cascades and the cell cycle, and thus impacts on disease. An exemplary area where this knowledge is rapidly expanding and contributing to novel therapies is cancer, where the Period genes have been identified as tumour suppressors. In more complex disorders, where aetiology remains controversial, interactions with the clockwork are only now starting to be appreciated.
Address Department of Clinical Neurosciences, University of Cambridge Metabolic Research Laboratories, Institute of Metabolic Science, Cambridge CB2 OQQ, UK. abr20@cam.ac.uk
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Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0962-8924 ISBN Medium
Area Expedition Conference
Notes PMID:19926479; PMCID:PMC2808409 Approved no
Call Number (up) IDA @ john @ Serial 133
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Author Schnitt, S.; Ruhtz, T.; Fischer, J.; Hölker, F.; Kyba, C.C.M.
Title Temperature stability of the sky quality meter Type Journal Article
Year 2013 Publication Sensors (Basel, Switzerland) Abbreviated Journal Sensors (Basel)
Volume 13 Issue 9 Pages 12166-12174
Keywords *Artifacts; Atmosphere/*analysis; Environmental Monitoring/*instrumentation; Equipment Design; Equipment Failure Analysis; Photometry/*instrumentation; Reproducibility of Results; Sensitivity and Specificity; Temperature; *Transducers; Sky Quality Meter; SQM
Abstract The stability of radiance measurements taken by the Sky Quality Meter (SQM)was tested under rapidly changing temperature conditions during exposure to a stable light field in the laboratory. The reported radiance was found to be negatively correlated with temperature, but remained within 7% of the initial reported radiance over a temperature range of -15 degrees C to 35 degrees C, and during temperature changes of -33 degrees C/h and +70 degrees C/h.This is smaller than the manufacturer's quoted unit-to-unit systematic uncertainty of 10%,indicating that the temperature compensation of the SQM is adequate under expected outdoor operating conditions.
Address Institute for Space Sciences, Freie Universitat Berlin, Carl-Heinrich-Becker-Weg 6-10, Berlin 12165, Germany. christopher.kyba@wew.fu-berlin.de
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Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 1424-8220 ISBN Medium
Area Expedition Conference
Notes PMID:24030682; PMCID:PMC3821345 Approved no
Call Number (up) IDA @ john @ Serial 194
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Author Chaves, I.; Pokorny, R.; Byrdin, M.; Hoang, N.; Ritz, T.; Brettel, K.; Essen, L.-O.; van der Horst, G.T.J.; Batschauer, A.; Ahmad, M.
Title The cryptochromes: blue light photoreceptors in plants and animals Type Journal Article
Year 2011 Publication Annual Review of Plant Biology Abbreviated Journal Annu Rev Plant Biol
Volume 62 Issue Pages 335-364
Keywords Adenosine Triphosphate/metabolism; Animals; Cryptochromes/chemistry/classification/*physiology; DNA Repair; Deoxyribodipyrimidine Photo-Lyase/chemistry/classification/physiology; Homing Behavior; Insects/physiology; *Light Signal Transduction; Magnetics; Mice; Oxidation-Reduction; Phosphorylation/physiology; Plants/*metabolism; blue light
Abstract Cryptochromes are flavoprotein photoreceptors first identified in Arabidopsis thaliana, where they play key roles in growth and development. Subsequently identified in prokaryotes, archaea, and many eukaryotes, cryptochromes function in the animal circadian clock and are proposed as magnetoreceptors in migratory birds. Cryptochromes are closely structurally related to photolyases, evolutionarily ancient flavoproteins that catalyze light-dependent DNA repair. Here, we review the structural, photochemical, and molecular properties of cry-DASH, plant, and animal cryptochromes in relation to biological signaling mechanisms and uncover common features that may contribute to better understanding the function of cryptochromes in diverse systems including in man.
Address Department of Genetics, Erasmus University Medical Center, 3000 CA Rotterdam, The Netherlands. i.chaves@erasmusmc.nl
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Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 1543-5008 ISBN Medium
Area Expedition Conference
Notes PMID:21526969 Approved no
Call Number (up) IDA @ john @ Serial 341
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