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Author Agarwal, N.; Srivastava, S.; Malik, S.; Rani, S.; Kumar, V.
Title Altered light conditions during spring: Effects on timing of migration and reproduction in migratory redheaded bunting (Emberiza bruniceps) Type Journal Article
Year 2015 Publication Biological Rhythm Research Abbreviated Journal Biological Rhythm Research
Volume 46 Issue 5 Pages 647-657
Keywords (up) Animals
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Language Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0929-1016 ISBN Medium
Area Expedition Conference
Notes Approved no
Call Number LoNNe @ christopher.kyba @ Serial 1166
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Author Zozaya, S.M.; Alford, R.A.; Schwarzkopf, L.
Title Invasive house geckos are more willing to use artificial lights than are native geckos: House geckos and artificial lights Type Journal Article
Year 2015 Publication Austral Ecology Abbreviated Journal Austral Ecology
Volume 40 Issue 8 Pages 982–987
Keywords (up) Animals
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Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 1442-9985 ISBN Medium
Area Expedition Conference
Notes Approved no
Call Number LoNNe @ christopher.kyba @ Serial 1209
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Author Wilson, A.-L.; Downs, C.T.
Title Light interference and melatonin affects digestion and glucocorticoid metabolites in striped mouse Type Journal Article
Year 2015 Publication Biological Rhythm Research Abbreviated Journal Biological Rhythm Research
Volume 46 Issue 5 Pages 929-939
Keywords (up) Animals
Abstract Animals secrete glucocorticoids to deal with daily stressors. Studies have found that supplemental melatonin decreases glucocorticoid metabolite levels in stressed animals. We determined the effect of light interference and supplemental melatonin on 1) body mass; 2) food intake; and 3) glucocorticoid metabolite levels of the striped mouse (Rhabdomys pumilio). Experiment was split into three phases: 8 L: 16 D; 8 L: 16 D with a 15 min light interruption every 4 h; and 8 L: 16 D with a 15 min light interruption every 4 h and melatonin (0.2 μg/ml) added to the water. Body mass was significantly different between phases with lowest body mass (89.17 ± 6.56 g) occurring during standard 8 L: 16 D. Light interference and melatonin significantly increased body mass. Light interference increased and melatonin decreased glucocorticoid metabolite levels. Light interference significantly increased and melatonin significantly decreased assimilation efficiencies possibly due to changes in energetic demands.
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Language Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0929-1016 ISBN Medium
Area Expedition Conference
Notes Approved no
Call Number LoNNe @ christopher.kyba @ Serial 1211
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Author Van Dycke, K.C.G.; Rodenburg, W.; van Oostrom, C.T.M.; van Kerkhof, L.W.M.; Pennings, J.L.A.; Roenneberg, T.; van Steeg, H.; van der Horst, G.T.J.
Title Chronically Alternating Light Cycles Increase Breast Cancer Risk in Mice Type Journal Article
Year 2015 Publication Current Biology : CB Abbreviated Journal Curr Biol
Volume 25 Issue 14 Pages 1932-1937
Keywords (up) Animals
Abstract Although epidemiological studies in shift workers and flight attendants have associated chronic circadian rhythm disturbance (CRD) with increased breast cancer risk, causal evidence for this association is lacking [1, 2]. Several scenarios have been proposed to contribute to the shift work-cancer connection: (1) internal desynchronization, (2) light at night (resulting in melatonin suppression), (3) sleep disruption, (4) lifestyle disturbances, and (5) decreased vitamin D levels due to lack of sunlight [3]. The confounders inherent in human field studies are less problematic in animal studies, which are therefore a good approach to assess the causal relation between circadian disturbance and cancer. However, the experimental conditions of many of these animal studies were far from the reality of human shift workers. For example, some involved xenografts (addressing tumor growth rather than cancer initiation and/or progression) [4, 5], chemically induced tumor models [6, 7], or continuous bright light exposure, which can lead to suppression of circadian rhythmicity [8, 9]. Here, we have exposed breast cancer-prone p53(R270H(c)/+)WAPCre conditional mutant mice (in a FVB genetic background) to chronic CRD by subjecting them to a weekly alternating light-dark (LD) cycle throughout their life. Animals exposed to the weekly LD inversions showed a decrease in tumor suppression. In addition, these animals showed an increase in body weight. Importantly, this study provides the first experimental proof that CRD increases breast cancer development. Finally, our data suggest internal desynchronization and sleep disturbance as mechanisms linking shift work with cancer development and obesity.
Address Department of Genetics, Center for Biomedical Genetics, Erasmus University Medical Center, Rotterdam 3000 CA, the Netherlands. Electronic address: g.vanderhorst@erasmusmc.nl
Corporate Author Thesis
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Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0960-9822 ISBN Medium
Area Expedition Conference
Notes PMID:26196479 Approved no
Call Number LoNNe @ christopher.kyba @ Serial 1221
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Author Hogan, M.K.; Kovalycsik, T.; Sun, Q.; Rajagopalan, S.; Nelson, R.J.
Title Combined effects of exposure to dim light at night and fine particulate matter on C3H/HeNHsd mice Type Journal Article
Year 2015 Publication Behavioural Brain Research Abbreviated Journal Behav Brain Res
Volume 294 Issue Pages 81-88
Keywords (up) Animals
Abstract Air and light pollution contribute to fetal abnormalities, increase prevalence of cancer, metabolic and cardiorespiratory diseases, and central nervous system (CNS) disorders. A component of air pollution, particulate matter, and the phenomenon of dim light at night (dLAN) both result in neuroinflammation, which has been implicated in several CNS disorders. The combinatorial role of these pollutants on health outcomes has not been assessed. Male C3H/HeNHsd mice, with intact melatonin production, were used to model humans exposed to circadian disruption by dLAN and contaminated environmental air. We hypothesized exposure to 2.5mum of particulate matter (PM2.5) and dLAN (5lx) combines to upregulate neuroinflammatory cytokine expression and alter hippocampal morphology compared to mice exposed to filtered air (FA) and housed under dark nights (LD). We also hypothesized that exposure to PM2.5 and dLAN provokes anxiety-like and depressive-like responses. For four weeks, four groups of mice were simultaneously exposed to ambient concentrated PM2.5 or FA and/or dLAN or LD. Following exposure, mice underwent several behavioral assays and hippocampi were collected for qPCR and morphological analyses. Our results are generally comparable to previous PM2.5 and dLAN reports conducted on mice and implicate PM2.5 and dLAN as potential factors contributing to depression and anxiety. Short-term exposure to PM2.5 and dLAN upregulated neuroinflammatory cytokines and altered CA1 hippocampal structural changes, as well as provoked depressive-like responses (anhedonia). However, combined, PM2.5 and dLAN exposure did not have additive effects, as hypothesized, suggesting a ceiling effect of neuroinflammation may exist in response to multiple pollutants.
Address Department of Neuroscience, Behavioral Neuroendocrinology Group, The Ohio State University, Columbus, OH 43210, USA
Corporate Author Thesis
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Language English Summary Language Original Title
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ISSN 0166-4328 ISBN Medium
Area Expedition Conference
Notes PMID:26235330 Approved no
Call Number LoNNe @ kyba @ Serial 1233
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