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Author Grubisic, M.; Van Grunsven, R.H.A.; Kyba, C.C.M.; Manfrin, A.; Hölker, F. openurl 
  Title Insect declines and agroecosystems: does light pollution matter? Type Journal Article
  Year 2018 Publication (up) Annals of Applied Biology Abbreviated Journal Ann. of Appl. Biol.  
  Volume 173 Issue 1 Pages 180-189  
  Keywords Animals; Ecology; Review  
  Abstract Drastic declines in insect populations, ‘Ecological Armageddon’, have recently gained increased attention in the scientific community, and are commonly considered to be the consequence of large‐scale factors such as land‐use changes, use of pesticides, climate change and habitat fragmentation. Artificial light at night (ALAN), a pervasive global change that strongly impacts insects, remains, however, infrequently recognised as a potential contributor to the observed declines. Here, we provide a summary of recent evidence of impacts of ALAN on insects and discuss how these impacts can drive declines in insect populations in light‐polluted areas. ALAN can increase overall environmental pressure on insect populations, and this is particularly important in agroecosystems where insect communities provide important ecosystem services (such as natural pest control, pollination, conservation of soil structure and fertility and nutrient cycling), and are already under considerable environmental pressure. We discuss how changes in insect populations driven by ALAN and ALAN itself may hinder these services to influence crop production and biodiversity in agricultural landscapes. Understanding the contribution of ALAN and other factors to the decline of insects is an important step towards mitigation and the recovery of the insect fauna in our landscapes. In future studies, the role of increased nocturnal illumination also needs to be examined as a possible causal factor of insect declines in the ongoing ‘Ecological Armageddon’, along with the more commonly examined factors. Given the large scale of agricultural land use and the potential of ALAN to indirectly and directly impact crop production and biodiversity, a better understanding of effects of ALAN in agroecosystems is urgently needed.  
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  Call Number GFZ @ kyba @ Serial 1939  
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Author Adams, J. url  openurl
  Title Duration of Light and Growth Type Journal Article
  Year 1924 Publication (up) Annals of Botany Abbreviated Journal  
  Volume 38 Issue 151 Pages 509-523  
  Keywords Plants  
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  Call Number GFZ @ kyba @ Serial 2391  
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Author Adams, J. url  openurl
  Title The Effect on Certain Plants of altering the Daily Period of Light Type Journal Article
  Year 1923 Publication (up) Annals of Botany Abbreviated Journal  
  Volume 37 Issue 145 Pages 75-94  
  Keywords Plants  
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  Notes Approved no  
  Call Number GFZ @ kyba @ Serial 2406  
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Author Lahti, T.; Merikanto, I.; Partonen, T. url  doi
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  Title Circadian clock disruptions and the risk of cancer Type Journal Article
  Year 2012 Publication (up) Annals of Medicine Abbreviated Journal Ann Med  
  Volume 44 Issue 8 Pages 847-853  
  Keywords Human Health; Cell Division; Chronobiology Disorders/*complications/genetics/*physiopathology; Circadian Clocks/*genetics; Humans; Neoplasms/*etiology; Work Schedule Tolerance/physiology  
  Abstract Disrupted circadian rhythms may lead to failures in the control of the cell division cycle and the subsequent malignant cell growth. In order to understand the pathogenesis of cancer more in detail, it is crucial to identify those mechanisms of action which contribute to the loss of control of the cell division cycle. This mini-review focuses on the recent findings concerning the links between the human circadian clock and cancer. Clinical implications concern not only feasible methods for the assessment of the circadian time of an individual or for the determination of the best time for administration of a drug of treatment, but also in the future genetic tests for screening and for planning treatment.  
  Address Department of Mental Health and Substance Abuse Services, National Institute for Health and Welfare, Helsinki, Finland  
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  ISSN 0785-3890 ISBN Medium  
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  Notes PMID:23072403 Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 513  
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Author Reiter, R.J.; Tan, D.-X.; Korkmaz, A.; Ma, S. url  doi
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  Title Obesity and metabolic syndrome: association with chronodisruption, sleep deprivation, and melatonin suppression Type Journal Article
  Year 2012 Publication (up) Annals of Medicine Abbreviated Journal Ann Med  
  Volume 44 Issue 6 Pages 564-577  
  Keywords Human Health; Adolescent; Adult; Animals; Child; Chronobiology Disorders/*epidemiology; Comorbidity; Disease Models, Animal; Humans; Light/adverse effects; Melatonin/*deficiency/physiology; Metabolic Syndrome X/*epidemiology; Mice; Obesity/*epidemiology; Rats; Sleep Deprivation/*epidemiology  
  Abstract Obesity has become an epidemic in industrialized and developing countries. In 30 years, unless serious changes are made, a majority of adults and many children will be classified as overweight or obese. Whereas fatness alone endangers physiological performance of even simple tasks, the associated co-morbidity of obesity including metabolic syndrome in all its manifestations is a far more critical problem. If the current trend continues as predicted, health care systems may be incapable of handling the myriad of obesity-related diseases. The financial costs, including those due to medical procedures, absenteeism from work, and reduced economic productivity, will jeopardize the financial well-being of industries. The current review summarizes the potential contributions of three processes that may be contributing to humans becoming progressively more overweight: circadian or chronodisruption, sleep deficiency, and melatonin suppression. Based on the information provided in this survey, life-style factors (independent of the availability of abundant calorie-rich foods) may aggravate weight gain. Both epidemiological and experimental data support associations between disrupted physiological rhythms, a reduction in adequate sleep, and light-at-night-induced suppression of an essential endogenously produced molecule, melatonin. The implication is that if these problems were corrected with life-style changes, body-weight could possibly be more easily controlled.  
  Address Department of Cellular and Structural Biology, UT Health Science Center, San Antonio, Texas, USA. reiter@uthscsa.edu  
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  ISSN 0785-3890 ISBN Medium  
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  Notes PMID:21668294 Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 523  
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