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Author Adams, J. url  openurl
  Title Duration of Light and Growth Type Journal Article
  Year 1924 Publication (up) Annals of Botany Abbreviated Journal  
  Volume 38 Issue 151 Pages 509-523  
  Keywords Plants  
  Abstract  
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  ISSN ISBN Medium  
  Area Expedition Conference  
  Notes Approved no  
  Call Number GFZ @ kyba @ Serial 2391  
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Author Adams, J. url  openurl
  Title The Effect on Certain Plants of altering the Daily Period of Light Type Journal Article
  Year 1923 Publication (up) Annals of Botany Abbreviated Journal  
  Volume 37 Issue 145 Pages 75-94  
  Keywords Plants  
  Abstract  
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  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
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  ISSN ISBN Medium  
  Area Expedition Conference  
  Notes Approved no  
  Call Number GFZ @ kyba @ Serial 2406  
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Author Lahti, T.; Merikanto, I.; Partonen, T. url  doi
openurl 
  Title Circadian clock disruptions and the risk of cancer Type Journal Article
  Year 2012 Publication (up) Annals of Medicine Abbreviated Journal Ann Med  
  Volume 44 Issue 8 Pages 847-853  
  Keywords Human Health; Cell Division; Chronobiology Disorders/*complications/genetics/*physiopathology; Circadian Clocks/*genetics; Humans; Neoplasms/*etiology; Work Schedule Tolerance/physiology  
  Abstract Disrupted circadian rhythms may lead to failures in the control of the cell division cycle and the subsequent malignant cell growth. In order to understand the pathogenesis of cancer more in detail, it is crucial to identify those mechanisms of action which contribute to the loss of control of the cell division cycle. This mini-review focuses on the recent findings concerning the links between the human circadian clock and cancer. Clinical implications concern not only feasible methods for the assessment of the circadian time of an individual or for the determination of the best time for administration of a drug of treatment, but also in the future genetic tests for screening and for planning treatment.  
  Address Department of Mental Health and Substance Abuse Services, National Institute for Health and Welfare, Helsinki, Finland  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
  Language English Summary Language Original Title  
  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN 0785-3890 ISBN Medium  
  Area Expedition Conference  
  Notes PMID:23072403 Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 513  
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Author Reiter, R.J.; Tan, D.-X.; Korkmaz, A.; Ma, S. url  doi
openurl 
  Title Obesity and metabolic syndrome: association with chronodisruption, sleep deprivation, and melatonin suppression Type Journal Article
  Year 2012 Publication (up) Annals of Medicine Abbreviated Journal Ann Med  
  Volume 44 Issue 6 Pages 564-577  
  Keywords Human Health; Adolescent; Adult; Animals; Child; Chronobiology Disorders/*epidemiology; Comorbidity; Disease Models, Animal; Humans; Light/adverse effects; Melatonin/*deficiency/physiology; Metabolic Syndrome X/*epidemiology; Mice; Obesity/*epidemiology; Rats; Sleep Deprivation/*epidemiology  
  Abstract Obesity has become an epidemic in industrialized and developing countries. In 30 years, unless serious changes are made, a majority of adults and many children will be classified as overweight or obese. Whereas fatness alone endangers physiological performance of even simple tasks, the associated co-morbidity of obesity including metabolic syndrome in all its manifestations is a far more critical problem. If the current trend continues as predicted, health care systems may be incapable of handling the myriad of obesity-related diseases. The financial costs, including those due to medical procedures, absenteeism from work, and reduced economic productivity, will jeopardize the financial well-being of industries. The current review summarizes the potential contributions of three processes that may be contributing to humans becoming progressively more overweight: circadian or chronodisruption, sleep deficiency, and melatonin suppression. Based on the information provided in this survey, life-style factors (independent of the availability of abundant calorie-rich foods) may aggravate weight gain. Both epidemiological and experimental data support associations between disrupted physiological rhythms, a reduction in adequate sleep, and light-at-night-induced suppression of an essential endogenously produced molecule, melatonin. The implication is that if these problems were corrected with life-style changes, body-weight could possibly be more easily controlled.  
  Address Department of Cellular and Structural Biology, UT Health Science Center, San Antonio, Texas, USA. reiter@uthscsa.edu  
  Corporate Author Thesis  
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  Language English Summary Language Original Title  
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  ISSN 0785-3890 ISBN Medium  
  Area Expedition Conference  
  Notes PMID:21668294 Approved no  
  Call Number LoNNe @ christopher.kyba @ Serial 523  
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Author Curtis, A.M.; FitzGerald, G.A. url  doi
openurl 
  Title Central and peripheral clocks in cardiovascular and metabolic function Type Journal Article
  Year 2006 Publication (up) Annals of Medicine Abbreviated Journal Ann Med  
  Volume 38 Issue 8 Pages 552-559  
  Keywords Human Health  
  Abstract  
  Address  
  Corporate Author Thesis  
  Publisher Place of Publication Editor  
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  Series Editor Series Title Abbreviated Series Title  
  Series Volume Series Issue Edition  
  ISSN 0785-3890 ISBN Medium  
  Area Expedition Conference  
  Notes Approved no  
  Call Number LoNNe @ kagoburian @ Serial 730  
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