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Author Chalkias, C.; Petrakis, M.; Psiloglou, B.; Lianou, M.
Title Modelling of light pollution in suburban areas using remotely sensed imagery and GIS Type Journal Article
Year 2006 Publication Journal of Environmental Management Abbreviated Journal J Environ Manage
Volume 79 Issue 1 Pages 57-63
Keywords Remote Sensing; Air Pollutants/*analysis; Cities; Environmental Monitoring/*methods; *Geographic Information Systems; Greece; Humans; *Light; Models, Theoretical; *Suburban Health
Abstract This paper describes a methodology for modelling light pollution using geographical information systems (GIS) and remote sensing (RS) technology. The proposed approach attempts to address the issue of environmental assessment in sensitive suburban areas. The modern way of life in developing countries is conductive to environmental degradation in urban and suburban areas. One specific parameter for this degradation is light pollution due to intense artificial night lighting. This paper aims to assess this parameter for the Athens metropolitan area, using modern analytical and data capturing technologies. For this purpose, night-time satellite images and analogue maps have been used in order to create the spatial database of the GIS for the study area. Using GIS advanced analytical functionality, visibility analysis was implemented. The outputs for this analysis are a series of maps reflecting direct and indirect light pollution around the city of Athens. Direct light pollution corresponds to optical contact with artificial night light sources, while indirect light pollution corresponds to optical contact with the sky glow above the city. Additionally, the assessment of light pollution in different periods allows for dynamic evaluation of the phenomenon. The case study demonstrates high levels of light pollution in Athens suburban areas and its increase over the last decade.
Address (up) Department of Geography, Harokopio University, El. Venizelou Str., Kalithea, 17671 Athens, Greece. xalkias@hua.gr
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0301-4797 ISBN Medium
Area Expedition Conference
Notes PMID:16171928 Approved no
Call Number LoNNe @ kagoburian @ Serial 729
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Author Haus, E.; Smolensky, M.
Title Biological clocks and shift work: circadian dysregulation and potential long-term effects Type Journal Article
Year 2006 Publication Cancer Causes & Control : CCC Abbreviated Journal Cancer Causes Control
Volume 17 Issue 4 Pages 489-500
Keywords Human Health; Adaptation, Physiological; Animals; Biological Clocks; Cardiovascular Abnormalities/etiology; Chronobiology Disorders/*complications/physiopathology; Chronobiology Phenomena; Humans; Neoplasms/etiology; Occupational Diseases/*etiology; Risk Factors; Suprachiasmatic Nucleus/physiopathology; *Work Schedule Tolerance
Abstract Long-term epidemiologic studies on large numbers of night and rotating shift workers have suggested an increase in the incidence of breast and colon cancer in these populations. These studies suffer from poor definition and quantification of the work schedules of the exposed subjects. Against this background, the pathophysiology of phase shift and phase adaptation is reviewed. A phase shift as experienced in night and rotating shift work involves desynchronization at the molecular level in the circadian oscillators in the central nervous tissue and in most peripheral tissues of the body. There is a change in the coordination between oscillators with transient loss of control by the master-oscillator (the Suprachiasmatic Nucleus, SCN) in the hypothalamus. The implications of the pathophysiology of phase shift are discussed for long-term health effects and for the design of ergonomic work schedules minimizing the adverse health effects upon the worker.
Address (up) Department of Laboratory Medicine & Pathology, University of Minnesota, Health Partners Medical Group, Regions Hospital, St. Paul, Minnesota 55101, USA. Erhard.X.Haus@Healthpartners.com
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0957-5243 ISBN Medium
Area Expedition Conference
Notes PMID:16596302 Approved no
Call Number LoNNe @ kagoburian @ Serial 760
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Author Jasser, S.A.; Blask, D.E.; Brainard, G.C.
Title Light during darkness and cancer: relationships in circadian photoreception and tumor biology Type Journal Article
Year 2006 Publication Cancer Causes & Control : CCC Abbreviated Journal Cancer Causes Control
Volume 17 Issue 4 Pages 515-523
Keywords Human Health; Animals; *Circadian Rhythm; *Darkness; Humans; *Light; Light Signal Transduction; Melatonin/physiology/secretion; Neoplasms/etiology/pathology/*physiopathology; Suprachiasmatic Nucleus/physiology
Abstract The relationship between circadian phototransduction and circadian-regulated processes is poorly understood. Melatonin, commonly a circadian phase marker, may play a direct role in a myriad of physiologic processes. The circadian rhythm for pineal melatonin secretion is regulated by the hypothalamic suprachiasmatic nucleus (SCN). Its neural source of light input is a unique subset of intrinsically photosensitive retinal ganglion cells expressing melanopsin, the primary circadian photopigment in rodents and primates. Action spectra of melatonin suppression by light have shown that light in the 446-477 nm range, distinct from the visual system's peak sensitivity, is optimal for stimulating the human circadian system. Breast cancer is the oncological disease entity whose relationship to circadian rhythm fluctuations has perhaps been most extensively studied. Empirical data has increasingly supported the hypothesis that higher risk of breast cancer in industrialized countries is partly due to increased exposure to light at night. Studies of tumor biology implicate melatonin as a potential mediator of this effect. Yet, causality between lifestyle factors and circadian tumor biology remains elusive and likely reflects significant variability with physiologic context. Continued rigorous empirical inquiry into the physiology and clinical implications of these habitual, integrated aspects of life is highly warranted at this time.
Address (up) Department of Neurology, Light Research Program, Thomas Jefferson University, 1025 Walnut Street, Suite 507, Philadelphia, PA 19107, USA. samar.jasser@jefferson.edu
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0957-5243 ISBN Medium
Area Expedition Conference
Notes PMID:16596305 Approved no
Call Number LoNNe @ kagoburian @ Serial 766
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Author Srinivasan, V.; Smits, M.; Spence, W.; Lowe, A.D.; Kayumov, L.; Pandi-Perumal, S.R.; Parry, B.; Cardinali, D.P.
Title Melatonin in mood disorders Type Journal Article
Year 2006 Publication The World Journal of Biological Psychiatry : the Official Journal of the World Federation of Societies of Biological Psychiatry Abbreviated Journal World J Biol Psychiatry
Volume 7 Issue 3 Pages 138-151
Keywords Human Health; Antidepressive Agents/therapeutic use; Biological Markers/blood; Bipolar Disorder/diagnosis/drug therapy/*physiopathology; Circadian Rhythm/drug effects/physiology; Depressive Disorder/diagnosis/drug therapy/*physiopathology; Depressive Disorder, Major/diagnosis/drug therapy/physiopathology; Humans; Melatonin/*blood/therapeutic use; Phototherapy; Seasonal Affective Disorder/diagnosis/physiopathology; Sleep Disorders, Circadian Rhythm/diagnosis/drug therapy/physiopathology; Treatment Outcome
Abstract The cyclic nature of depressive illness, the diurnal variations in its symptomatology and the existence of disturbed sleep-wake and core body temperature rhythms, all suggest that dysfunction of the circadian time keeping system may underlie the pathophysiology of depression. As a rhythm-regulating factor, the study of melatonin in various depressive illnesses has gained attention. Melatonin can be both a 'state marker' and a 'trait marker' of mood disorders. Measurement of melatonin either in saliva or plasma, or of its main metabolite 6-sulfatoxymelatonin in urine, have documented significant alterations in melatonin secretion in depressive patients during the acute phase of illness. Not only the levels but also the timing of melatonin secretion is altered in bipolar affective disorder and in patients with seasonal affective disorder (SAD). A phase delay of melatonin secretion takes place in SAD, as well as changes in the onset, duration and offset of melatonin secretion. Bright light treatment, that suppresses melatonin production, is effective in treating bipolar affective disorder and SAD, winter type. This review discusses the role of melatonin in the pathophysiology of bipolar disorder and SAD.
Address (up) Department of Physiology, School of Medical Sciences, University Sains Malaysia, Kubang Kerian, Kota Bharu, Kelantan
Corporate Author Thesis
Publisher Place of Publication Editor
Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 1562-2975 ISBN Medium
Area Expedition Conference
Notes PMID:16861139 Approved no
Call Number LoNNe @ kagoburian @ Serial 816
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Author Stoffels, W.W.
Title Gravity's pull on arc lamp efficiency Type Journal Article
Year 2006 Publication Europhysics News Abbreviated Journal Europhysics News
Volume 37 Issue 6 Pages 35-38
Keywords Lighting; physics; plasma; gravity; lighting technology; lighting physics; metal halide; hypergravity; microgravity
Abstract (none)
Address (up) Eindhoven University of Technology, PO Box 513, 5600 MB Eindhoven, Netherlands; w.w.stoffels(at)tue.nl
Corporate Author Thesis
Publisher Place of Publication Editor
Language Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0531-7479 ISBN Medium
Area Expedition Conference
Notes Approved no
Call Number IDA @ john @ Serial 1367
Permanent link to this record