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Author Fisher, N.A.
Title Reclaiming the „Dark Sky“. Type Journal Article
Year 2006 Publication Electrical Contracting Products Abbreviated Journal
Volume (up) 9 Issue 6 Pages 30–31
Keywords Energy; Lighting
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Notes Approved no
Call Number LoNNe @ kagoburian @ Serial 1036
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Author Faraji, H.; MacLean, W.J.
Title CCD noise removal in digital images Type Journal Article
Year 2006 Publication IEEE Transactions on Image Processing Abbreviated Journal IEEE Trans. on Image Process.
Volume (up) 15 Issue 9 Pages 2676-2685
Keywords Remote Sensing
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ISSN 1057-7149 ISBN Medium
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Notes Approved no
Call Number LoNNe @ kagoburian @ Serial 935
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Author Laaksonen, J.; Laaksonen, T.; Itämies, J.; Rytkönen, S.; Välimäki, P.
Title A new efficient bait-trap model for Lepidoptera surveys – the “ Oulu ” model. Type Journal Article
Year 2006 Publication Entomologica Fennica Abbreviated Journal
Volume (up) 17 Issue Pages 153–160
Keywords Animals
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Notes Approved no
Call Number LoNNe @ kagoburian @ Serial 607
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Author Bullough, J.D.; Rea, M.S.; Figueiro, M.G.
Title Of mice and women: light as a circadian stimulus in breast cancer research Type Journal Article
Year 2006 Publication Cancer Causes & Control : CCC Abbreviated Journal Cancer Causes Control
Volume (up) 17 Issue 4 Pages 375-383
Keywords Human Health; Animals; Breast Neoplasms/*physiopathology; *Circadian Rhythm; *Disease Models, Animal; Female; Humans; *Light; Light Signal Transduction; Mammary Neoplasms, Animal/*physiopathology; Melatonin/metabolism; Mice; Muridae/metabolism
Abstract OBJECTIVE: Nocturnal rodents are frequently used as models in human breast cancer research, but these species have very different visual and circadian systems and, therefore, very different responses to optical radiation or, informally, light. Because of the impact of light on the circadian system and because recent evidence suggests that cancer risk might be related to circadian disruption, it is becoming increasingly clear that optical radiation must be properly characterized for both nocturnal rodents and diurnal humans to make significant progress in unraveling links between circadian disruption and breast cancer. In this paper, we propose a quantitative framework for comparing radiometric and photometric quantities in human and rodent studies. METHODS: We reviewed published research on light as a circadian stimulus for humans and rodents. Both suppression of nocturnal melatonin and phase shifting were examined as outcome measures for the circadian system. RESULTS: The data were used to develop quantitative comparisons regarding the absolute and spectral sensitivity for the circadian systems of humans and nocturnal rodents. CONCLUSIONS: Two models of circadian phototransduction, for mouse and humans, have been published providing spectral sensitivities for these two species. Despite some methodological variations among the studies reviewed, the circadian systems of nocturnal rodents are approximately 10,000 times more sensitive to optical radiation than that of humans. Circadian effectiveness of different sources for both humans and nocturnal rodents are offered together with a scale relating their absolute sensitivities. Instruments calibrated in terms of conventional photometric units (e.g., lux) will not accurately characterize the circadian stimulus for either humans or rodents.
Address Lighting Research Center, Rensselaer Polytechnic Institute, 21 Union Street, Troy, NY 12180, USA. bulloj@rpi.edu
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Language English Summary Language Original Title
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ISSN 0957-5243 ISBN Medium
Area Expedition Conference
Notes PMID:16596289 Approved no
Call Number LoNNe @ kagoburian @ Serial 726
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Author Filipski, E.; Li, X.M.; Levi, F.
Title Disruption of circadian coordination and malignant growth Type Journal Article
Year 2006 Publication Cancer Causes & Control : CCC Abbreviated Journal Cancer Causes Control
Volume (up) 17 Issue 4 Pages 509-514
Keywords Human Health; Animals; Biological Clocks; Body Temperature; Cell Cycle Proteins; Cell Line, Tumor; Chronobiology Disorders/*complications/physiopathology; Circadian Rhythm; Corticosterone/blood; DNA-Binding Proteins/metabolism; Jet Lag Syndrome/complications/physiopathology; Lymphocyte Count; Mice; Neoplasm Transplantation; Nuclear Proteins/metabolism; Nuclear Receptor Subfamily 1, Group D, Member 1; Osteosarcoma/*pathology/physiopathology; Pancreatic Neoplasms/*pathology/physiopathology; Period Circadian Proteins; Receptors, Cytoplasmic and Nuclear/metabolism; Suprachiasmatic Nucleus/physiopathology; Transcription Factors/metabolism
Abstract Altered circadian rhythms predicted for poor survival in patients with metastatic colorectal or breast cancer. An increased incidence of cancers has been reported in flying attendants and in women working predominantly at night. To explore the contribution of circadian structure to tumor growth we ablated the 24-h rest-activity cycle and markedly altered the rhythms in body temperature, serum corticosterone and lymphocyte count in mice by complete stereotaxic destruction of the suprachiasmatic nuclei (SCN) or by subjecting the mice to experimental chronic jet-lag. Such disruption of circadian coordination significantly accelerated malignant growth in two transplantable tumor models, Glasgow osteosarcoma and Pancreatic adenocarcinoma. The mRNA expression of clock genes per2 and reverb-alpha in controls displayed significant circadian rhythms in the liver (Cosinor, p=0.006 and p=0.003, respectively) and in the tumor (p=0.04 and p<0.001, respectively). Both rhythms were suppressed in the liver and in the tumor of jet lagged mice. This functional disturbance of molecular clock resulted in down regulation of p53 and overexpression of c-Myc, two effects which may favor cancer growth. CONCLUSIONS: These results indicate that circadian system could play an important role in malignant growth control. This should be taken into consideration in cancer prevention and therapy.
Address INSERM E 354 Cancer Chronotherapeutics, Hopital Paul Brousse, Villejuif, France. filipski@vjf.inserm.fr
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Language English Summary Language Original Title
Series Editor Series Title Abbreviated Series Title
Series Volume Series Issue Edition
ISSN 0957-5243 ISBN Medium
Area Expedition Conference
Notes PMID:16596304 Approved no
Call Number LoNNe @ kagoburian @ Serial 748
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